Name: Platelet-Vessel Wall Interactions (en Inglés)
Price: 506.54 PEN
Availability: InStock
Author: Pittilo, R. Michael ; Machin, Samuel J.
ISBN: 9781447114574

portada Platelet-Vessel Wall Interactions (en Inglés)

Formato

Libro Físico

Editorial

Springer

Autor

Pittilo, R. Michael ; Machin, Samuel J.

Idioma

Inglés

N° páginas

198

Encuadernación

Tapa Blanda

Dimensiones

24.4 x 17.0 x 1.2 cm

Peso

0.35 kg.

ISBN13

9781447114574

Categorías

Fisiologiía
Enfermedades cardiovasculares
Hematología

Platelet-Vessel Wall Interactions (en Inglés)

Pittilo, R. Michael ; Machin, Samuel J. (Autor) · Springer · Tapa Blanda

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Origen: Estados Unidos (Costos de importación incluídos en el precio)

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Reseña del libro "Platelet-Vessel Wall Interactions (en Inglés)"

The publication of Platelet-Vessel Wall Interactions, the second monograph in the Bloomsbury Series in Clinical Science, is particu- larly welcome as its appearance signifies the further development of the Series and its potential for the future. The theme of this monograph is the pathophysiology of atherosclerosis, a topic that symbolises the aim of the Series, namely to highlight the important interfaces between basic medical science and clinical practice. Our congratulations to the Editors and contributors. London, December 1987 lack Tinker Preface In the Western world, atherosclerosis causes more illness and death than any other disease. Despite its devastating effects, the pathogenesis of the disease remains a matter for hypothesis and con- jecture. This monograph owes its conception to a programme of work directed towards understanding the basic pathophysiology of atherosclerosis. The circulatory system is lined by vascular endothelium which has a central role in maintaining the integrity of the vessel wall and prevent- ing thrombosis. The natural equilibrium existing between normal en- dothelium which supports blood flow, and platelets which serve to re- pair damaged endothelium, is explored in the first two chapters. Atherosclerosis developing as a response to endothelial injury is one hypothesis which has stimulated widespread interest, and re- search has largely been directed towards finding the injurious agent.